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Animal studies have failed to reveal evidence of teratogenicity or embryofetal toxicity when administered in doses up to 28 to 98 times MHRD (based on AUC comparisons).

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To test the therapeutic effect of HO-1 activator in vivo, IMQ-induced psoriasis mouse models were established through the topical application of 5% IMQ cream.

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Thus our results demonstrating the effects of imiquimod treatment on NFκB activation and secretion of the cytokines IL-6 and CCL20 in primary keratinocytes indicated an activity beyond TLR7 or TLR8.

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Prior presentation: Much of this work was presented as the author's MSc Thesis in Clinical Dermatology; permission for use granted by St.

Although an endogenous immune response to cancer is observed in preclinical models and patients, this response is ineffective, because tumors develop multiple resistance mechanisms, including local immune suppression, induction of tolerance, and systemic dysfunction in T-cell signaling.

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The complementary method of claim 60, wherein the lesion-directed cytodestruction therapy is administered no more than about 14 days prior to the commencement of the application of the field-directed immunotherapy.

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Evaluation of relatives at risk: Because of the need for surveillance for complications of NBCCS (medulloblastoma in children; jaw cysts and BCCs in adults) and the need to avoid sun exposure, clarification of the genetic status of at-risk relatives, including children, is appropriate.

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